Neuropeptide Y increases 4-aminopyridine-sensitive transient outward potassium current in rat ventricular myocytes

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Neuropeptide Y increases 4-aminopyridine-sensitive transient outward potassium current in rat ventricular myocytes.

1. The modulation of 4-aminopyridine sensitive transient outward potassium current (4-AP I(to)) by neuropeptide Y (NPY) (100 nM) in rat ventricular myocytes was examined using the whole cell voltage-clamp technique. 2. Continuous exposure to NPY (100 nM) for 3 - 6 h significantly increased 4-AP I(to) density. The stimulation of 4-AP I(to) density by NPY was concentration-dependent (EC(50)=10 nM...

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Use-dependent features of 4-aminopyridine block of transient outward current in rat ventricular myocytes.

The inhibition of the calcium-independent transient outward current (Ito) by the widely used blocker 4-aminopyridine (4-AP) was studied in adult Wistar rat ventricular myocytes, enzymatically isolated and voltage-clamped in the whole cell configuration using patch-clamp pipettes. 4-AP at 1 mmol/l concentration caused complete steady-state block of Ito at resting or hyperpolarized voltages. The ...

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Effect of acidosis on transient outward potassium current in isolated rat ventricular myocytes.

The effect of acidosis on the transient outward K(+) current (I(to)) of rat ventricular myocytes has been investigated using the perforated patch-clamp technique. When the holding potential was -80 mV, depolarizing pulses to potentials positive to -20 mV activated I(to) in subepicardial cells but activated little I(to) in subendocardial cells. Exposure to an acid solution (pH 6.5) had no signif...

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The effect of catecholamine depletion (induced by prior treatment with reserpine) was studied in Wistar rat ventricular myocytes using whole cell voltage-clamp methods. Two calcium-independent outward currents, the transient outward potassium current (I(to)) and the sustained outward potassium current (I(sus)), were measured. Reserpine treatment decreased tissue norepinephrine content by 97%. A...

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Two functionally distinct 4-aminopyridine-sensitive outward K+ currents in rat atrial myocytes

In the experiments here, the detailed kinetic properties of the Ca(2+)-independent, depolarization-activated outward currents (Iout) in enzymatically dispersed adult rat atrial myocytes were studied. Although there is only slight attenuation of peak Iout during brief (100 ms) voltage steps, substantial decay is evident during long (10 s) depolarizations. The analyses here reveal that current in...

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ژورنال

عنوان ژورنال: British Journal of Pharmacology

سال: 2002

ISSN: 0007-1188

DOI: 10.1038/sj.bjp.0704800